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Inhibitors of GH signal transduction pathways (post-receptor) Given that GH activates JAK-STAT, PI3K/AKT/mTOR and MAPK signaling, agents which target components of these signaling pathways would be expected to modulate pathway-specific GH effects
Using this computational prediction method, we found that Gclc fl/fl MZB exhibited increased glucose catabolism compared to Gclc fl/fl FoB (Fig
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