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Figure 3 below summarizes these processes, highlighting the interplay between bacterial infection, immune mediators (e.g., TNF-, IL-6, transforming growth factor-beta [TGF-], IL-10), and cellular actors (e.g., neutrophils, M1/M2 macrophages, fibroblasts, keratinocytes, endothelial cells) during wound healing [43, 48]

Findings from multiple animal studies indicate expedited healing of stomach ulcers and decreased tissue damage in stressful settings [4]

Measurements were obtained from the right lobe of the liver through the intercostal spaces, with a patient lying in the dorsal decubitus position and the right arm in maximal abduction

Cardiovasc Diabetol 20:205