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Collectively, these studies imply mutant ALS proteins produce inflammatory cytokines, disturb redox homeostasis, and increase neuroinflammation and toxicity

Once absorbed, intracellular esterases remove the acetyl group to release free glutathione

Ischemic heart disease including myocardial infarction and chronic ischemia could potentially benefit from the peptide's cardioprotective effects, enhanced coronary angiogenesis, and anti-arrhythmic properties demonstrated in animal models

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