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doi: 10.1016/j.yebeh.2018.12.010 13

Metabolic changes included a reduction of the usage of muscle glycogen (i.e., its carbohydrate stores) during low-intensity exercise (consistent with an increase in lipid utilization) and streamlined glucose metabolism and mitochondrial flux during high-intensity exercise, leading to reduced anaerobic ATP production (which is what leads to lactic acid build-up in muscles)

These directly damage hepatocyte membranes and mitochondria while depleting GSH, impairing detoxification capacity