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The cells were treated with metformin, and various cellular functions were assessed, including proliferation, apoptosis, migration, and epithelial-mesenchymal transition (EMT) status ( In summary, metformin disrupts the normal proliferation process of glioma cells and triggers apoptosis by activating the AMPK/mTOR signaling pathway, cell cycle arrest, mitochondria-dependent apoptosis pathway, inhibition of tumor cell metabolism, oxidative stress, and DNA damage, which provides a new idea for the treatment of glioma

Programmed cell death in diabetic kidney disease: mechanisms and therapeutic targeting

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Glutathione injections for fertility can improve the reproductive microenvironment