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Blocking GSH synthesis in combination with JMF4073 depletes dTTP/dCTP pools to eradicate T315I-Bcr-Abl leukemia We hypothesize that reducing the GSH level to increase replication stress can render T315I-Bcr-Abl leukemia more susceptible to JMF4073 in vivo
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Studies (101,102) have indicated that the GSH/GSSG redox status serves as a vital indicator of tumor programmed cell death, consistently associating programmed cell death with a decrease in the GSH/GSSG ratio
2-Todo medicamento deve ser mantido fora do alcance de crianas
It promotes the inhibition of tyrosinase activity by blocking the interaction of melanocytes and keratinocytes via the inhibition of the plasminogen/plasmin system
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