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It also downregulates key extracellular matrix regulators, resulting in improved liver function and histopathology
in the other outcome, the injured tissue continues to deteriorate, and the mechanism of the persisting damage involves gliosis with the loss of support of neurotrophic factors, persistent inflammatory receptor activation, and changes in microglia and astrocytes that continue to release harmful cytokines, which promote neuronal death and axonal injury
More studies are needed to evaluate how glutathione deficiency triggers diseases and to determine adequate dosing
Chest 153 , 851862 (2018)
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