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This review will explore the neurological basis of ADHD and examine how disruptions in the GBA, through immune, neural, and microbial pathways, may influence brain function and contribute to ADHD pathophysiology, highlighting current findings, proposed mechanisms, and potential clinical implications

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Interestingly, the glucose levels in rats receiving exendin-4 via the intestine also increased and comparison of the AUCs, taking into account the difference in quantity of peptide administered suggests a relative biopotency of 7%

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