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Depolarization of pericytes can also result from neuronal transmitters like NA, ATP, and Ang2 binding to their corresponding receptors on pericytes, as well as from astrocytic AA entering pericytes [102, 103]

Identifying the functionally important renal substrates of DPP4 is necessary to understand and predict the long-term effects of DPP4 inhibition on the kidney in patients with diabetes

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