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7 In patients with T2DM, the release of incretin hormones is physiologically insufficient, thereby compromising the ability to adequately secrete insulin and also execute other physiologic processes required to meet the metabolic requirements to maintain normoglycemia
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Insulin sensitivity improvements manifest through multiple metabolic markers
As underling mechanism of the interaction between afferent and efferent renal nerves our previous report [24] has shown that activation of afferent renal nerves leads to activation of efferent renal sympathetic nerves response to GLP-1, possibly via central neural reflex in normal rats
Further Reading For additional peer-reviewed research on this topic, see: PubMed research on KPV anti-inflammatory activity
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