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Thus, although some of the damage in this experimental model of colitis reflects a direct erosive effect of TNBS and ethanol, their combined administration to rats reflects the in vivo operation of the damaging mechanisms elicited by TNBS, namely GSH depletion and ROS generation, indicating that ethanol facilitates the access of TNBS to the cell interior ensuring TNBS-induced GSH depletion and ROS generation

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This interaction, however, may not be its own individual communication pathway, but rather the BBB is an interface of communication between the gut microbiota, blood, and the brain

This action slows down gastric emptying, reduces appetite, and enhances feelings of satiety

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