Traditionally treatment of lcFAODs consists of prevention of catabolism, LCT restriction, and supplementation of MCT [143, 145, 146]
Several important issues, however, remain to be addressed
J Pharmacol Exp Ther 311:99108 Clancy RM, Levartovsky D, Leszczynska-Piziak J, Yegudin J, Abramson SB (1994) Nitric oxide reacts with intracellular glutathione and activates the hexose monophosphate shunt in human neutrophils: evidence for S-nitrosoglutathione as a bioactive intermediary
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At Cymbiotika, we favor the liposomal approach for glutathione because it directly addresses the bioavailability challenge