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With advancements in microbiome research, gut microbiota have been identified as pivotal regulators of host metabolism and immunity, modulating host redox homeostasis through metabolite secretion, epithelial barrier repair, and immunomodulation [6,7,8]
While it is not possible to roll back the clock, deeper understanding of the biological pathways and mechanisms through which adverse childhood experiences produce a lifelong vulnerability to altered mitochondrial metabolism and the related pathways can provide a path for compensatory plasticity toward more positive health directions
In 2012, Mena et al
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