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Even though lixisenatide and exenatide are both GLP-1R agonists, they differ in brain penetration, duration of action, and pharmacologic properties which likely explains the discrepancy in Parkinsons trial outcomes

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The fibrotic process is a dysregulated wound-healing response, driven by the persistent activation of fibroblasts (or myofibroblasts), which results in the deposition of excessive extracellular matrix (ECM) proteins such as collagen [4], a key feature of pathological tissue remodelling in fibrotic disorders [5,6,7,8]