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Similarly, genetically engineering the overexpression of tyrosine hydroxylase is associated with higher levels of phosphorylated tyrosine hydroxylase, extracellular dopamine, reactive metabolites, and intermediates such as H 2 O 2 and 3,4-dihydroxyphenylacetaldehyde (DOPAL) [5] Taken together, levels of tyrosine hydroxylase and dopamine increase immediately following methamphetamine exposure but tend to decrease afterward [5, 71, 97, 98, 101, 102]