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Our systematic characterization of semaglutides signalling mechanisms in the hindbrain reveals the intracellular signalling architecture through which semaglutide engages cAMP and calcium to regulate body weight, providing avenues for improving obesity therapeutics.
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Saturated fatty acids induce c-Src clustering within membrane subdomains, leading to JNK activation
Pathophysiology of fibroid disease: angiogenesis and regulation of smooth muscle proliferation
Pleiotropic effects of thiazolidinediones: taking a look beyond antidiabetic activity
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