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aureus ) [47], prolong the inflammatory phase by stimulating continuous immune activation

Its proposed mechanism is to stimulate lipolysis (the breakdown of stored fat) and to inhibit lipogenesis (the formation of new fat), apparently through -adrenergic-related pathways, while unlike full-length GH not raising IGF-1 and not impairing glucose tolerance [1] [5]

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It's important to note that while these preclinical findings are compelling, human clinical trials remain limited