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Together, they engage two separate hormonal systems that both regulate body weight creating an additive, and potentially synergistic, effect on appetite suppression and calorie reduction

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CD8 + T cells (a type of cytotoxic T lymphocyte) expressing NKG2D bind to NKG2D ligands produced by HFECs to form MHC-I antigen complexes and thus become effector cells

The potential mechanism for this effect includes the deposition of collagen through the ERK1/2 signaling pathway, re-epithelialization and glandular tissue formation [3]