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Obesity disrupts adipose plasticity specifically by suppressing 3-adrenergic receptor (AR)-mediated white adipose tissue (WAT) browning via inflammatory pathways

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Additionally, clinical trials with long-acting dual or triple agonists targeting the glucagon receptor have demonstrated a marked suppressive effect on circulating amino-acids in both humans and animals [66, 67]

Blocking GABA A receptors enhanced network activity and resulted in an increase in glutamatergic excitatory inputs (Fig