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33,45,46 There are studies illustrating that GST interaction with STAT3 can inhibit the STAT3-signaling pathway, curb aberrant cell-cycle progression, and decrease cell proliferation

[4] That selectivity is part of why the drug made sense for HIV-associated lipodystrophy specifically, where the clinical problem is central/visceral fat accumulation

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2004 Apr;63(4):641-6.

In contrast, 1919 cells appear to preferentially channel glucose carbons into the pentose phosphate pathway rather than serine and glycine synthesis for nucleotide synthesis, as indicated by higher levels of m + 5 isotopologues (Fig