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In preclinical models it accelerates mucosal tissue repair, supports intestinal lining integrity, reduces markers of intestinal permeability, and promotes angiogenesis in gastrointestinal tissue

In addition, LA stimulated glucose uptake upon translocation and regulation of the intrinsic activity of GLUT4, an effect that is likely mediated by p38 MAPK

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Similar content being viewed by others Introduction Inflammation is a well-known cardiovascular risk factor acting as a facilitator for atherosclerosis and atherosclerotic plaque rupture