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[ 3 H]BPC157 was rapidly metabolized into a variety of small peptide fragments in vivo , thus forming single amino acids that entered normal amino acid metabolism and excretion pathways

Several studies have shown that overexpression of CDC42 from neural stem cell-derived exosomes and Tp53-induced glycolysis and apoptosis regulator (TIGAR) significantly suppresses lipid peroxidation and ferroptosis, thereby promoting neurological recovery

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