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Our results suggest that both increased S -glutathionylation processes and suppressed de-glutathionylation reactions contribute to hepatic S -glutathionylation in NAFLD
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In vitro, measurements revealed that the increase in cortical BMC was due to an increased cross-sectional bone area, whereas the cortical volumetric BMD was unchanged
GSSG prevented TNF-induced activation of IKK-, an upstream kinase in the NF-B signaling pathway, by inducing IKK- glutathionylation (IKK--SSG formation)
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