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Hypoxia and intense exercise promote inflammatory activation and oxidative stress that contribute to mitochondrial dysfunction, often via disturbed fusionfission balance and accumulation of damaged mitochondria/mtDNA ( Mitochondrial remodeling in response to Meldonium is not necessarily benign: increased mitochondrial turnover or exercise-induced stress can elevate ROS and mtDNA vulnerability, particularly with impaired antioxidant defenses or DNA repair (30, 31)

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Five studies involving 825 exposed pregnancies and 592,714 comparator pregnancies evaluated first-trimester exposure and major congenital malformations

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To view a copy of this licence, visit About this article Cite this article Liu, S., Kanamori, Y., Ohta, Y