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When dopamine signaling related to reward anticipation is reduced, observations may include: Less preoccupation with food-related cues Reduced reward-seeking behavior In some cases, reduced responsiveness to other rewarding stimuli This is the context in which GLP-1based compounds intersect with reward neurobiology

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In this review, we discuss how mitochondrial dysfunction contributes to the manifestation of PTSD symptoms and how this may even increase PTSD susceptibility

GLP-1 drugs might lead people to shun alcohol entirely: or the effect may be more nuanced