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Exendin-4 exposure also led to a significant reduction in the level of reactive oxygen species (ROS) in Control shRNA islets exposed to the ER stress drug thapsigargin, but this effect was no longer present in SPHKAP shRNA-transduced islets, which presented with generally higher ROS levels than the corresponding control islets (Fig

Thus, an increase in NRF2 activity, reflected by induction of NRF2 target genes, likely represent an important mechanism mediating the protection seen in GCLM KO mice

Importantly, the medications long half-life guarantees consistent, durable impacts, minimizing dosing frequency

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