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The basis for this resistance is the weakening of the red cell membrane (the erythrocyte is the host cell for the parasite) such that it cannot sustain the parasitic life cycle long enough for productive growth.
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Excess ROS and RNS disrupt these proteins and their linkage to actin, widening the paracellular space and enabling translocation of luminal antigens, toxins, and microbes
Nutritional Biochemistry (Academic Press, San Diego, 1999)
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