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In the lungs, after entering in type II alveolar cells, SARS-CoV-2 infected cells become defective for surfactant production (Ghati et al., 2021) and release cytokines, like IL-8 among others, which in turn activate alveolar macrophages to release IL-1, IL-6, and TNF-, that induce natural killer and dendritic cell differentiation and macrophage M1 polarization, enhancing the proinflammatory response with increased vasodilation causing neutrophil and activated T cell influx from the capillaries into the alveolus, all leading to the cytokine storm (Carcaterra and Caruso, 2021

Systematic assessment of animal studies revealed a potential association of several molecular pathways, including AMPK-, SIRT1, FDX, PI3 K/AKT, endothelial adhesion molecules (VCAM, ICAM, and E-selectin), STAR, and CYP17 A1, with the therapeutic effects of GLP-1 RAs and SGLT2is in PCOS

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I dosed at 240mcg per week first two weeks