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In addition to the current mechanism of the effects of the MeDiet on the response to oxidation and inflammation, another candidate and potential neuroprotective mechanism might be associated with the roles mediated by this dietary pattern in regulating and/or protecting the gut microbiota composition of the gut-brain axis (7, 114)

It consists of the tripeptide glycyl-L-histidyl-L-lysine complexed with a divalent copper ion, forming a high-affinity chelate that facilitates controlled copper delivery within biological systems

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