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The kidney is the primary site of B 12 concentration and storage in rodents, from where it is released for use by other organs upon systemic deficiency 27 ,29,30,31

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Crucially, the researchers noted that the S-A-GSH molecule was rapidly deacetylated once inside the body, proving that the acetyl shield successfully protected the compound through the digestive tract and then efficiently released the intact, active glutathione directly into the intracellular environment where it is needed to combat oxidative stress

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