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This process may initiate the inappropriate incorporation of selenocysteine (SeCys) and selenomethionine (SeMet) into proteins, as well as impair their function, which may in turn lead to the development of oxidative stress, i.e., a state in which the oxidizing potential increases to the level that threatens the stability of cellular structures [14]

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These reactive species can damage surrounding tissues and DNA, promoting mutations and genomic instability

These include, for example, hepatitis, tumors or metastases in the liver