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That said, the current work greatly advances our understanding of how bacterial signalling in the gut can both induce and also curtail intestinal inflammation, and reveals an exciting role for the gut hormone GLP-1 as a potential therapeutic target in the regulation of inflammatory diseases of the intestine
Furthermore, an increase in the intragastric pH, observed during GLP-1 RAs therapy, may also alter the absorption of medications (48)
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Mechanistically, JTC801 disrupts lysosomal pH, leading to cholesterol accumulation and activation of SREBF2, a master transcriptional regulator of sterol biosynthesis
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