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For example, in tendon fibroblast cells, BPC-157 dramatically increases phosphorylation (activation) of FAK and paxillin, leading to more cell migration and survival at injury sites

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D: Neurodegenerativediseases (e.g., Alzheimers disease, Parkinsons disease, frontotemporal dementia, amyotrophic lateral sclerosis)

They overlap with trial findings but reflect a different exposure pattern (lower per-dose, daily, longer duration)

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