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This protection is especially important for men over 50, when oxidative stress increases dramatically

These defects are speculated to be caused by cellular stresses generated under this control condition, whereas the wild-type strain might have adapted to the stresses through a basal level of autophagy

However, major uncertainties remain regarding environmentally relevant exposure levels, long-term human effects, and the translational relevance of current experimental findings

doi:10.1016/j.ajog.2019.09.007

We note that although the chondroprotective effects of metformin was clearly attenuated in Nrf2 KO mice, micro-CT analysis revealed some variability in trabecular bone parameters, including occasional increases in subchondral trabecular bone amounts in Nrf2 KO OA mice (Fig