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Lipotoxicity is associated with chronic cellular stresses (endoplasmic reticulum stress, oxidative stress) and the impairment of protective mechanisms such as autophagy, promoting liver injury and the enrichment of danger signals (damage-associated molecular patterns or DAMPs, extracellular vesicles, hepatocyte senescence, etc.) leading to MASLD progression [51,52,53]

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Babies at birth are naturally exposed to the hyperoxic challenge due to the transition from the hypoxic intrauterine environment (pO 2 of 2025 mmHg) to extrauterine life (pO 2 of 100 mmHg)