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Finding the roots of adult neurogenesis
Hu Y, Benya RV, Carroll RE, Diamond AM
Recently, nonsteroidal antiinflammatory drugs have been shown to partially impair an equilibrium between biological functioning and degradation of aldosterone due to involvement of renal UGT2B7 in both the glucuronidation of aldosterone (deactivation) and the glucuronidation of NSAIDs (Knights et al., 2009)
This approach maintains the natural IGFBP-binding architecture and pituitary feedback regulation, avoiding the risks associated with direct exogenous IGF-1 receptor stimulation [19]
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