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In contrast, GGCT knockdown led to a significant increase in the extracellular acidification rate (ECAR) (Fig
These phenotypes include not only the contractile VSMCs marked by MYH11, -SMA, Calponin, TAGLN, and MYOCD [23, 24], but also synthetic VSMCs marked by RUNX2, MSX2, SOX9, COL2A1, and SP7 [25], macrophage-like VSMCs expressing LGALS3, CD68, TNF-, and IL-1 [26, 27], foam cell-like VSMCs marked by UCP1 and ADIPSIN [28, 29], mesenchymal-like VSMCs marked by SCA1, CD34, and CD44 [30], osteogenic-like VSMCs marked by RUNX2, MSX2, SOX9, and OSX [31], myofibroblast-like VSMCs expressing PDGFR [6], fibroblast-like VSMCs expressing LUM, BGN, DCN, FN1, COL1A1, and COL1A2 [32, 33], aging VSMCs marker by SA--gal and SASP [34], and inflammatory VSMCs marked by NF-B, MCP1, IL6, CCL5, and CXCL10 [35, 36]
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