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Many neuroprotective effects remain to be causally associated with anti-inflammatory mechanisms, and some Alzheimers disease mouse models did not respond to semaglutide or tirzepatide, emphasizing that not all neural benefits are consistent across studies
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315 The results showed that, in the OA rat model, liraglutide could activate the PKA/CREB signaling pathway and inhibit the inflammatory response through this pathway, thereby alleviating OA symptoms
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