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Thus, the reduction in colonic GSH in our model is most appropriately interpreted as reflecting both impaired glutathione synthesis and increased oxidative demand, rather than biosynthetic impairment alone

Originally only described for the reduction of GSH-mixed disulfides, the monothiol reaction mechanism may also be functional for the reduction of certain protein disulfides, e.g., mammalian RNR and engineered GFP variants [36,37]

BPC-157 has demonstrated analgesic (pain-relieving) effects in various studies

Melittin alleviates sepsis-induced acute kidney injury by promoting GPX4 expression to inhibit ferroptosis