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Our results uncover a new noncanonical function of PKM2 as a transcriptional coactivator of Nrf2, which cooperates with its metabolic function to divert glucose flux into the pentose phosphate pathway 55 and thereby generates sufficient GSH for ROS detoxification

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Researchers selecting between the two typically start with the question being asked: is the study modeling a systemic growth factor response, or a localized repair response triggered at a specific site

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