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In animal models of asthma, GLP-1RA suppress IL-33 release, limit ILC2 proliferation, and decrease IL-5 and IL-13 production, indicating downregulation of Th2-driven inflammation [7]
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The conclusion was that, based on animal studies and epidemiological studies, acetaminophen exposure during infancy was a greater risk factor than in utero exposure for ASD
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Macrophage cytotoxicity against schistosomula of Schistosoma mansoni involves arginine-dependent production of reactive nitrogen intermediates
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