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In renal injury models, extracellular vesicles isolated from hypoxia-treated human proximal tubular epithelial cells induce ferroptosis in previously untreated recipient tubular cells
In summary, the reprogramming of hepatocytes associated with a profibrotic milieu results in loss of mature parenchymal cells, characterized by loss of the master hepatocyte transcription factor HNF4 (158, 212) and upregulation of the developmental Notch, Hippo (213), and Hedgehog (214) signaling pathways
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