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In a homocysteine-induced cognitive impairment model, an independent risk factor for AD, betaine alleviated cognitive impairment by inhibiting the NLRP3/caspase-1/GSDMD pathway in an m 6 A-YTHDF2-dependent manner, thereby suppressing microglial pyroptosis (187)

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Then, correcting deficiencies helps restore cellular function and improve mitochondrial energy production, support lean muscle mass, and create a more resilient metabolism that can better maintain weight and appetite regulation after coming off a GLP-1