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Mechanistic studies indicate that AKT-mTOR signaling can reduce cytoplasmic malonyl-CoA levels by inhibiting ACACA, thereby influencing CPT1 activity and promoting fatty acid entry into mitochondria for -oxidation [173, 174]
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Body composition (DEXA or similar) only useful with a long enough window to detect change
10.1016/S0140-6736(85)91481-3 Ohtani Y, Endo F, Matsuda I: Carnitine deficiency and hyperammonemia associated with valproic acid therapy
We next tested the converse prediction: that promoting glutathione reduction would cause ER stress
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