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The functional consequence in animal models is synergistic rather than additive GH output: GHRH-R priming increases the pool of releasable GH vesicles, and GHS-R1a co-stimulation then drives a larger amplitude pulse than either receptor produces alone

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(2006) studied the effect of NAC (50 and 100 mg/kg for 8 days) on PTZ-induced seizures in mice (60 mg/kg via intraperitoneal injection)