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It significantly reduced oxidative stress and injury in persons with diabetic ketoacidosis without an increase in blood sugar
Like glutamate, GABA is not metabolized in the synapse after release but is instead transported back into the GABA neuron or, more commonly, into adjacent astrocytes, where it is converted to glutamine
These products are not approved for human use and operate outside the legal compounding system
Additionally, mean monthly headache days were significantly reduced in the exenatide group compared to placebo, further supporting its therapeutic potential [99]
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