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In addition, GLP-1, but not GIP, inhibits glucagon secretion, and together these actions form the basis for the use of GLP-1 receptor agonists (GLP-1RAs) in T2DM therapy [6]

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However, excessive BCACs produced by the breakdown of branched-chain amino acids can interfere with normal mitochondrial function and cause energy metabolism disorders [11]

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Our cell viability assay revealed that knockdown of SLC25A39 inhibited the growth of CRC cells compared to controls, whereas upregulation of SLC25A39 expression resulted in the opposite effect (Fig