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In the late stages of infection, various cell deaths, including ferroptosis, promote inflammatory cytokine release, exacerbating immune and inflammatory system dysfunction and causing associated damage that ultimately leads to COVID-19 multi-organ complications (Sun et al., 2022)
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Hasanpour Z, Javanmard SH, Gharaaty M, Sadeghi M
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On the other hand, FoB rely on OXPHOS to a lower extent: Gclc -deficient FoB lose CI and CII functionality, but these cells increase glycolysis to replenish their ATP pool to the extent that they persist in Gclc fl/fl Mb1 Cre + hosts
