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Hada, M
The information presented herein is accurate to the best of the author's knowledge at the time of publication
The pathophysiology of AKI in sepsis is complex and multifactorial, encompassing intrarenal hemodynamic changes, endothelial dysfunction, infiltration of inflammatory cells within the renal parenchyma, and obstruction of renal tubules by necrotic cells and debris (112,113)
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Regular injections can provide sustained benefits
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