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Clinically, GLP-1RAs like semaglutide induce sweet hyposensitivity and reduce preference for hyperpalatable foods, an effect mediated by changes in endogenous GLP-1 secretion and synaptic plasticity in reward circuits (166)

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This is the rationale for FINE-ONE attempting to translate a well-established T2D paradigm into the long-neglected space of DKD in patients with type 1 diabetes

Given the complex interplay between metabolism, bone development, cell differentiation, and the gut microbiota during growth, these pharmacological interventions must be applied with caution and supported by robust longitudinal evidence